Skoue/shows Worcester 2013 La Rhone Legacy LR 1090 Jnr en Groot Kampioen Bul Jnr Interras Kamp Bul Eienaar: La Rhone Limousins La Rhone John Deere LR 0977 Snr Kampioen Limousin Bul Eienaar: La Rhone Limousins La Rhone Miracle LR 1143 Jnr Kampioen Limousin Vers Eienaar: La Rhone Limousins La Rhone Garland LR 0739 Snr en Groot Kampioen Limousin Koei Eienaar: La Rhone Limousins Ermelo 2012 61
Skoue/shows Standerton 2012
Bien Donné Agri KAAP Week 2012 Bien donné 2013 Jan Braai bespreek die voordele van braai met goeie vleis Noulin Blom, blokman van Tomi s Meatpackers Aubrey Martin, (abattoir- en slaghuiskonsultant) verduidelik die voordele van n Limousinkarkas en vleiskwaliteit Bennie Nothnagel - Vleisaankoper vir Spar in die Wes-Kaap Dr Gerhard Neetling - Algemene Bestuurder van die Rooivleisabattoirvereniging aan die woord
Hoërskool Bekker Bekzema Limousinstoet Die afgelope jaar is die diere vir verskeie aktiwiteite gebruik, waarvan die hoogtepunt die Nasionale Jeugskou was. Die skou is gehou gedurende die September skoolvakansie in Clanwilliam. Baie dankie aan Sas Oosthuizen wat sy diere tot ons beskikking gestel het en ook die diere vervoer het. In die Groepsklasse by die Juniors het ons leerlinge wat deel was van die Gauteng span n algehele derde plek behaal. Individueel het ons n 5de en 7de plek behaal. Die diere is ook gebruik om die leerlinge te assesseer ten opsigte van die volgende aspekte in beesboerdery: Merk- en Registrasieprosedure, brand van horings, die volg van n ent-en doseerprogram. Verder is die beeste gebruik vir die opleiding van die jeugskouleerlinge in die hantering en versorging van die beeste. Verder vir die jaar beplan ons weer die skoudag wat aangebied sal word. Ons wil graag ook in die derde kwartaal n Junior Beoordelaarskursus aangebied vir ons Graad 11 Landboubestuurspraktykleerlinge. Die ongediertes het weer toegeslaan en ons het 2 kalwers verloor. Daar is 15 vroulike diere gesinchroniseer en van hulle is 10 met kunsmatige inseminasie bevrug. Die ander is weer by die bul geplaas en ons sal binnekort laat dragtigheidsondersoeke doen. Tans staan die kudde op 29 diere. Mnr. Alex Wehmeyer wat vir die kudde verantwoordelik was is aangestel as die hoof by Morgenzon Landbou Akademie in Mpumalanga. Namens al die betrokke partye wil ons vir hom baie gelukwens en sterkte toewens met sy taak. Baie dankie Alex vir al jou insette en harde werk. Ons hoop om binnekort te weet wie in sy pos aangestel is. Namens myself en die skool baie dankie aan die genootskap en al die individue wat insette gelewer het die afgelope jaar. Bekkergroete Brian Callaghan Adjunkhoof 66
Skoue/shows Jeugskou 2012 Limousin SA ondersteun trots Jeugskoue landswyd 67
Limousin tour to France 2011 Paul van Biljon 68 With Paul and JC under construction the other tour members had their hands full! Mobility for long periods was sometimes a challenge for both Paul after a neck operation and JC busy healing from a fractured pelvis due to a car accident. And on top of this everything had to happen within limited time frames: riding on trains, flying on plains! Being on time was important for every day s planning! Communication was a big problem and the French people were not always as helpful as one had hoped for! Luckily all went well in the end with our president Sas Oosthuizen and his communication skills, in charge of the French liaison. We landed in Amsterdam and from there flew to Charles de Gaulle Airport outside Paris, France where we had to catch the last train of the day to Limoges to be in time for our visit to the Limousin Headquarters in France. Luckily for Thea and Marie, who had some international traveling experience, we managed to find the right train just in time. (continue on p71)
(continued from p68) At Limoges we visited the bull test station where Limousin bulls are tested before they can qualify to be presented and sold as breeding bulls. From Limoges we went to Clermont- Ferrand where we attended the National Limousin show (Sommet d elevage) and this was a huge show to remember! With around one million registered Limousins in France, one can imagine that the standard of Limousins entered in the show would be very high. In some of the classes to be judged, there were more than 30 animals in a group! Eggers; Sas and Marie Oosthuizen; Paul and Ronel van Biljon; Chris and Thea Gerber; JC, Roneel and Elize Steenkamp with Tippie; Johann Claassen; Willie Britz and Rudi Hinze. Many thanks to all of you! Until next time! We visited some Limousin farms (with some of the studs being in a family for 3 generations!) during the few days at the show where we also spent time with the president of the French Limousin Society on his farm. From Clermont-Ferrand we travelled to Paris for a few days - getting to know the French people better! This was surely a tour to remember! Our tour members were: Kobus and Maryn van Tonder; Johan and Amien 71
Tour to France 2011 72
Muscle up with Limousin F O R H E AV Y W E I G H T P R O F I T S! Dr Wayne Pitchford, office@limousin.co.au Questions and Answers about the Limousin Muscling Gene What is the function of the myostatin gene? A normally functioning myostatin gene inhibits muscle growth. A mutation of this gene reduces its powers to switch off the multiplication of muscle fibres which can result in an increase of the size and number of muscle fibres. There are nine known mutations of the myostatin gene of which six are loss of function mutations meaning they inactivate the myostatin gene causing muscular hypertrophy (larger muscle fibres) and hyperplasia (more muscle fibres). The best known mutation is the 11 pair deletion nt821 which has a high frequency in the Belgian Blue breed causing the typical double muscling effect. This mutation also occurs in low frequencies in other breeds such as the Angus, Murray Grey and South Devon breeds. Other loss of function mutations include the Q204 mutation in Charolais and the C313Y mutation in Piedmontese. The loss of function myostatin mutations are associated with high levels of calving difficulty and lowered fertility and longevity. What is different about the Limousin muscling gene? The mutation known as F94L which has a high frequency in the Limousin breed is one of three missence mutations which does not cause a loss of function of the myostatin gene. It appears not to cause an increase in the size of the muscle fibres but does cause an increase in the number of muscle fibres. It is not associated with high levels of calving difficulty or lowered fertility and longevity. What are the effects of the Limousin muscling gene? The research conducted by the Adelaide University group led by Dr Wayne Pitchford has clearly documented the effects of the Limousin muscling gene as shown below. How big is the effect if I use a Limousin bull over cows of another breed? Most Limousin bulls carry two copies of the Limousin muscling gene so each of the progeny will get one copy of the Limousin muscling gene. Animals with one copy of the gene are likely to exhibit about half the differences shown above. A Limousin cross calf is expected to have about 9.5% more silverside, 9.5% more eye muscle area 4% more retail beef yield, with a 3% increase in tenderness of the loin muscle and 5.5% more tender silverside. 73
Why still Vaccinate Brucellosis causes losses through abortions, reduced milk production, higher somatical cell counts, extended intercalving periods, fewer replacement heifers and a decrease in value of a breeding herd. Brucellosis is a serious disease which is transmitted to humans; thus becoming a public health hazard. Bovine brucellosis will impede international trade in cattle and their by-products 7, 10, 13. The stigma attached to the disease unfortunately impedes transparency and the effective control thereof 2. Open communication on the status of herds will curb its spreading through timeous pro-active sharpening up of their biosecurity by adjacent enterprises. Brucellosis is a serious zoonosis Bovine brucellosis is highly contagious. Humans become contaminated by contact with aborted fetuses, postpartum uterus excretions, ingestion of unpasteurized milk or drops of afterbirth fluid from positive animals that come in contact with the human s eye mucous membranes. Brucellosis in humans is known as Undulant Fever or Malta Fever, which is easily confused with diseases like flu, and for that reason often not correctly diagnosed. It is because the symptons are generally non-specific. Symptoms include: chronic joint and muscular pain, intermittent fever, fatigue, depression, night sweat attacks, orgitis in males and abortions in pregnant females. People with chronic brucellosis exhibit amongst others backache, hepatitis, valvulitis (inflammation of the heart valve) and meningitis. The disease inflicts a severe blow to quality of life. What does the law say about vaccination against brucellosis? The Directorate of Animal Health launched the Bovine Brucellosis Scheme in 1979. In accordance with the Animal Disease Act (Act35/ 1984) all heifers between age 4-8 months MUST be injected with S19 vaccine at least once in their lifetime. (Reg.no.G0101 Act 36/ 1947) especially important where the prevalence is high and control in the surrounding areas poor 8, 9. Booster doses with S19 vaccine or the use of it in mature cows is illegal, because it will cause false positive diagnostic tests 1, 3, 5, 8. It is disappointing that currently probably less than 15% of the heifers that are born each year are ultimately injected with S19 inoculant. One vaccination does NOT guarantee 100% protection With a single S19 vaccination between 4-8 months only 65% - 75% of heifers are protected against abortion 3, 5, 9. Re-vaccination with RB-51 (Reg. no. G3056 Wet36/1947) of heifers before first mating does not interfere with diagnostic serological tests 11, 15, is permissible and is strongly recommended to improve protection against abortion 4, 6 from 2 months till the first S19 enting at age 4-8 months. No absolute protection is guaranteed by any of the brucellosis vaccines however often administered 3, 8, 10. What it amounts to is that vaccination only forms part as an aid in the total strategy against bovine brucellosis. 74 HEIFERS AND COWS AGAINST BOVINE BRUCELLOSIS? Willem Schultheiss and Adel de Haast, willem.schultheiss@ceva.com In spite of vaccination, exposure to the virulent field strain will cause a positive response. Vaccination with S19 before age 8 months and booster doses with RB-51 does NOT deprive the heifer of her ability to launch a humeral immune response after being challenged by a virulent brucellosis field strain. Such a response involves the development of antibodies that will make the serological tests positive in spite of vaccination by any vaccine. The presence of antibodies does not necessarily mean that the animal is contaminated at the time of testing, but indicates previous exposure 9. Vaccination only helps to restrict production losses and the risk of abortion due to brucellosis. An overwhelming sustained contamination of field strain will overcome vaccine induced protection and abortion may follow. It is therefore clear that vaccination alone will not clear the risk of the consequences of brucellosis contamination. Prevention of exposure to contamination from outside and spreading thereof within a herd requires the same sustained dedication as with vaccination 3, 5. When is the best time to vaccinate multiparous cows in a positive herd with RB-51? Multiparous cows must be in an increasing level of energy balance and non-pregnant 3, 5. Vaccinate mature cows annually between 30 40 days after calving, but not earlier [will not generate good immunity in cows that are still experiencing a negative energy balance (NEB)], and also not later (can impair conception). The immunity system will determine how the body will react to a vaccination. Intake of a balanced diet is imperative for an effective immune system 2. What to do if the herd is positive: on-farm biosecurity Recommendations of the State Veterinarian must be followed 1 and may vary, depending on the percentage of positive cattle. In each case cattle may only leave the farm with a Red Cross permit for slaughter purposes. Brucella bacteria can survive on dry land for up to 1-2 months, in aborted fetuses in the shade for up to 8 months, water polluted with faeces to 8 months and 3-4 months in manure 3, 5, 7. For the time that positive cattle remain in the herd, rigid biosecurity measures should be put in place to prevent the disease from spreading further 1, 10. The following are amongst others important: Separate the positive and negative cattle at least shortly before -, during and 40 days after calving and also during milking where the positive cows are the last to be milked before the milk machine is disinfected. Vaccination of mature cattle as recommended by the State Veterinarian should be adhered to. RB-51 has been used with success in this regard 6, 17, 18. It will not render the serological test positive and hence does not counteract the diagnostic capacity. Avoid calving camps on grass since it promotes the concentration of contamination. Allow cows to calve separately on cement with bedding. Disinfect the environment after each calving.
Colostrum of contaminated cows should not be given to calves of clean cows 3. Destroy contaminated material and incinerate immediately, before dogs, cats, jackals or other scavengers that have preyed on placentae or aborted fetuses spread the bacteria through their excretion 2, 16. Effective fly control is imperative. Flies that have been in contact with contaminated material will contaminate the cow via the eye mucous membranes. Since it takes only 1,000 brucellosis bacteria (10 3 organisms) for a cow to become contaminated and the placenta fluid contains 10 9 organisms per ml, it is logical that it is a trifle for flies to transmit one millionth to other cattle via the eye mucous membranes. It is not uncommon for flies to be a major problem on farms especially in the calf raising facility.the first vaccination of an already positive calf at 4 months will not keep the already settled infection in the calf from causing an abortion with the first pregnancy. Fly control is therefore a necessity in the prevention of spreading of brucellosis on the farm. Use separate equipment (eg wheelbarrows, spades) for positive and negative cattle. Regular cleaning of feeding and water troughs if contamination takes place through birth fluids. References: 1. Anon. 1998 Brucellosis in Cattle. DAFF 2. Anon 2010 LHPG Expert Meeting on Brucellosis, Harrismith. 3. Bishop G.C., Bosman P.P. Herr S. 1994 Bovine Brucellosis. In: Infectious Diseases of Southern Africa. Ed. Coetzer J.A.W. et. al. Oxford Univ. Press. 4. Cutler S.J. et.al. 2005 Brucellosis New Aspects of an old Disease. Journal of Applied Microbiology 98:1270-1281 5. Godfroid J., Bosman P.P., Herr S., Bischop G.C. 2004 Bovine brucellosis. In: Coetzer (Ed) Infectious Diseases of Livestock. Vol 3: 1507-1527. Oxford Univ Press. 6. Lord V.R. et. al. 1998 Field study of vaccination of cattle with Brucella abortus strains RB51 and 19 under high and low disease prevalence. AJVR Vol59, No8 7. McDermott J.J., Arimi S.M. 2002 Brucellosis in sub-saharan Africa: epidemiology, control and impact. Veterinary Microbiology 90: 111-134 8. Moriyón I. et.al. 2004 rough vaccines in animal brucellosis: Structural and genetic basis and present status. Vet. Res. 35:1-38 9. Olsen S.C., Tatum F. 2010 Bovine Brucellosis.Vet Clin Food Anim 26:15-17 10. Olsen S.C., Stoffregen W.S. 2005 Essential role of vaccines in brucellosis control and eradication programs for livestock. www. future-drugs.com. ISSN1476-0584 11. Samartino L.E. et. al. 2000 Use of Brucella abortus vaccine strain RB51 in pregnant cows after calf hood vaccination with strain 19 in Argentina. Preventative Veterinary Medicine 45: 193-199 12. Sauret J.M. Vilissova N 2002 Human Brucellosis. J. Am. Board Fam. Pract.; 15:401-406 13. Seleem M.N. et. al. 2010 Brucellosis: A re-emerging zoonosis. Veterinary Microbiology 140: 392-398 14. Stevens M.G., Olsen S.C. Cheville N.F. 1995 Comparative analysis of immune responses in cattle vaccinated with Brucella abortus strain 19 of strain RB51. Veterinary Immunology and Immunopathology 44:223-235 15. Stevens M.G., Olsen S.C., Palmer M.V., Cheville N.F. 1997 Brucella abortus Strain RB51: A new Brucellosis Vaccine for Cattle. Compendium of Continuing Education Vol 19 No 6. 16. Davis D.S. et.al. 1988 Interspecific Transmission of Brucella abortus from experimentally infected Coyotes (Canis latrans) to parturient Cattle. Jnl of Wildlife Diseases 24(3) 533-537 17. Martins H. et. al. 2009 Eradication of Bovine Brucellosis in the Azores, Portugal Outcome of a 5 year programme (2002-2007) based on test-and-slaugheter and RB-51 vaccination. Prev. Vet. Med 90: 80-89 18. Sanz C et al. 2010 Mass vaccination as a compulsory tool in the control of a severe outbreak of bovine brucellosis due to Brucella abortus in Extremadura, Spain. In press: Prev. Vet. Med, doi:10.1016/j.prevetmed.2101.08.003
Limousin Veilings & Boeredae Veilingsuitslae 2012 VEILING DATUM HOOGSTE BULLE HOOGSTE KOEIE HOOGSTE VERSE BUL gemid KOEI gemid VERS gemid VERGESIGTE 15/02/2012 90,000.00 31,833.33 16,000.00 14,000.00 CLAASSEN FAMILIE TRUST 27/03/2012 38,000.00 18,833.33 15,000.00 14,142.86 DEVLAN 04/04/2012 65,000.00 55,000.00 15,000.00 RENLO 18/04/2012 30,000.00 24,250.00 18,000.00 13,969.00 SENTRALE BULTELERS GROEP 01/08/2012 34,000.00 22,846.00 18,000.00 12,875.00 13,000.00 10,466.00 BOSVELD VEILING 07/08/2012 28,000.00 21,000.00 14,500.00 11,357.14 13,500.00 11,562.50 LA RHONE 01/09/2012 130,000.00 51,615.79 130,000.00 45,304.35 CROPWELL 27/09/2012 122,000.00 30,500.00 ELITE VERSVEILING 28/09/2012 137,500.00 91,666.00 56,000.00 28,062.00 DEVLAN 14/11/2012 140,000.00 43,538.00 30,000.00 23,900.00 42,000.00 26,375.00 Veilingsuitslae 2013 VEILING DATUM HOOGSTE BULLE HOOGSTE KOEIE HOOGSTE VERSE BUL gemid KOEI gemid VERS gemid SAJAMA 15/02/2013 52,000.00 28,590.00 35,000.00 28,083.00 40,000.00 19,519.00 VERGESIGTE 20/02/2013 20,000.00 19,333.00 42,000.00 15,250.00 DEVLAN 04/03/2013 45,000.00 38,333.00 20,000.00 17,500.00 20,000.00 17,400.00 Vergesigte 77
renlo veiling Devlan veiling Alle ras bulveiling Ermelo 79